Living with Alzheimers

I have lived with two people that have alzheimer’s and it is consuming care that needs to be provided. For me, I had another family members help. .I had another family member who helped me with the care of the person who had alzheimer’s. There are many people that are not so lucky and they have to care for their parent or family member. All by themselves all the time and it is can be very consuming. And interrupt and interrupt many things that the person is working on.

I lived with my mom until they until they died, another i’m living with right now, they have minor dementia. It’s no secret that, as you get around forty five to 50 years old, you start to not be as sharp and as fast as you was in your twenties. What I keep reading is there’s many different things that contribute to alzheimer’s. And this article here talks about it plainly, that for sure, Alzheimer’s disease is happening, even though you don’t see any symptoms. This means leads me to believe that even though i’m only fifty six years old, I probably have alzheimer’s disease, it i’s just not symptomatic right now. Then, there’s many things that can be causing this.It could partially be mitochondrial problems inside my cells.What happens is mitochondrial dna starts to get deletions.So they call it mitochondrial dna deletions. These deletions of the d n a cannot be repaired, and they basically are the root cause of a lot of problems with nervous system.Muscle wasting also partially alzheimer’s related.

hippocampus:

It converts short-term memories into long-term memories by organizing, storing and retrieving memories within your brain. Your hippocampus also helps you learn more about your environment (spatial memory), so you’re aware of what’s around you, as well as remembering what words to say (verbal memory).

https://www.mdpi.com/1467-3045/48/3/334

Aβ/tau accumulates for years before overt cognitive symptoms, implying early circuit-level and neurotransmitter system dysfunction that precedes dementia onset.
Recent research has evidenced that tau and amyloid affect distinct brain circuits from early stages: tau pathology, especially in the hippocampus, gives rise to memory deficits, while Aβ deposition in the amygdala is associated with early emotional disturbances such as anxiety and fear, both contributing to the clinical picture through synergistic neuroinflammatory effects and brain malfunction

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